Pathological linkage study of SARS-CoV-2 N protein targeting COX-2 overexpression in transgenic mouse model

  • Funded by The Structural Genomics Consortium (SGC)
  • Total publications:5 publications

Grant number: C-0031

Grant search

Key facts

  • Disease

    COVID-19
  • start year

    -99
  • Known Financial Commitments (USD)

    $0
  • Funder

    The Structural Genomics Consortium (SGC)
  • Principal Investigator

    Yinxiong Li, Johnny Kim
  • Research Location

    China, Germany
  • Lead Research Institution

    Guangzhou Institutes of Biomedicine and Health, Max Planck Institute for Heart and Lung Research
  • Research Priority Alignment

    N/A
  • Research Category

    Pathogen: natural history, transmission and diagnostics

  • Research Subcategory

    Pathogen morphology, shedding & natural history

  • Special Interest Tags

    N/A

  • Study Type

    Unspecified

  • Clinical Trial Details

    N/A

  • Broad Policy Alignment

    Pending

  • Age Group

    Not Applicable

  • Vulnerable Population

    Not applicable

  • Occupations of Interest

    Not applicable

Abstract

Publicationslinked via Europe PMC

Hepatic DKK1-driven steatosis is CD36 dependent.

Neutralizing serum amyloid a protects against sinusoidal endothelial cell damage and platelet aggregation during acetaminophen-induced liver injury.

COVID-19-associated liver injury: Clinical characteristics, pathophysiological mechanisms and treatment management.

Modeling MEN1 with Patient-Origin iPSCs Reveals GLP-1R Mediated Hypersecretion of Insulin.

Efficiently generate functional hepatic cells from human pluripotent stem cells by complete small-molecule strategy.