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Elucidation of human platelet function activation mechanism by SARS-CoV-2 and search for therapeutic method

Grant number: 21K09044

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Key facts

  • Disease

    COVID-19

  • Start & end year

    2021
    2024

  • Known Financial Commitments (USD)

    $37,440

  • Funder

    KAKENHI

  • Principal Investigator

    Shuji Kawamoto Unspecified

  • Research Location

    Japan

  • Lead Research Institution

    Kyoto University

  • Research Priority Alignment

    N/A

  • Research Category

    Clinical characterisation and management

  • Research Subcategory

    Disease pathogenesis

  • Special Interest Tags

    N/A

  • Study Type

    Unspecified

  • Clinical Trial Details

    N/A

  • Broad Policy Alignment

    Pending

  • Age Group

    Not Applicable

  • Vulnerable Population

    Not applicable

  • Occupations of Interest

    Not applicable

Abstract

2 Publications linked via Europe PMC

Last Updated:4 days ago

View all publications at Europe PMC

SARS-CoV-2 Omicron variant spike protein maintains the ability to bind and activate human platelets: Comparison with the wild-type.

Variant-derived SARS-CoV-2 spike protein does not directly cause platelet activation or hypercoagulability.