SBIR Phase II: Stress Pathway Inhibition To Prevent COVID-19 Infection (COVID-19)

  • Funded by National Science Foundation (NSF)
  • Total publications:0 publications

Grant number: 2150149

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Key facts

  • Disease

    COVID-19
  • Start & end year

    2022
    2024
  • Known Financial Commitments (USD)

    $990,000
  • Funder

    National Science Foundation (NSF)
  • Principal Investigator

    Donald Davidson
  • Research Location

    United States of America
  • Lead Research Institution

    CREATIVE BIOTHERAPEUTICS LLC
  • Research Priority Alignment

    N/A
  • Research Category

    Therapeutics research, development and implementation

  • Research Subcategory

    Pre-clinical studies

  • Special Interest Tags

    N/A

  • Study Type

    Unspecified

  • Clinical Trial Details

    N/A

  • Broad Policy Alignment

    Pending

  • Age Group

    Not Applicable

  • Vulnerable Population

    Not applicable

  • Occupations of Interest

    Not applicable

Abstract

The broader impact/commercial potential of this Small Business Innovation Research (SBIR) Phase II project advance the treatment of COVID-19. A viral infection like that leading to COVID-19 creates stress on cells similar to cancer, obesity, diabetes and aging. This project advances a single non-toxic injection for critical patients to reduce cellular stress, block virus infection, and increase survival. In addition, this biologic therapy is also effective against the SARS-CoV-2 mutations, Ebola, and Influenza A. This has the potential to transform treatment for virus infections and cancer therapy.

This Small Business Innovation Research (SBIR) Phase II project advances a treatment to end COVID-19 by blocking the SARS-CoV-2 receptors on lung cells. The project advances discoveries that a survival protein, GRP78, is essential for virus infectivity and that an associated inhibitor can prevent infection. The project optimizes methods to use a lead GRP78 inhibitor to block spike proteins (SPs) of SARS-CoV-2 and mutations, as well as other virus receptor binding domains (RBDs) from binding to receptors and to lung cells. It is anticipated that the efficacy of the lead GRP78 inhibitor to block whole virus SARS-CoV-2, Ebola, and Influenza A viruses' infection on lung cells will exceed 99%.

This award reflects NSF's statutory mission and has been deemed worthy of support through evaluation using the Foundation's intellectual merit and broader impacts review criteria.